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In white adipose tissue of obese, insulin-resistant and aged mammals, NNMT is dramatically upregulated and because it consumes both the universal methyl donor (SAM) and the NAD precursor (nicotinamide), its hyperactivity simultaneously depletes the cellular methylation reserve and the NAD-salvage pool [1,2]

This results in a higher concentration of the medication being delivered to the body, providing targeted local or systemic effects

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Minor effects may include mild bruising or cool sensations during infusionthese resolve quickly and are rare